Good question, so why don’t you ask that cardiologists? When the pericardium becomes inflamed, the amount of fluid between the heart and the pericardium increases. This squeezes the heart and restricts its action and in his case, may have caused the AF/Flutter.
I am concerned of the possible side effects of the beta blocker and wonder if there are some natural beta blockers that could be used instead.
Patients with Sick Sinus Syndrome may develop alternating too slow and/or too fast heart rhythms, also known as tachy-brady syndrome:
Like in your case, often patients don’t have any symptoms, or if not, may present with fatigue, dizziness, palpitations, and (near) fainting. Sick sinus syndrome may be aggravated by some drugs that include Beta Blockers.
I’m wondering does having had pectus excavatum increase my risk of afib later on and also since my dad just got afib at 72 does that increase my risk . The last time I went to the doc for a regular checkup the dr felt my heart and didn’t say there was a murmur or anything wrong, however I’m just wondering how genetic is afib . I read one article that said 2/3 of ppl who had lone afib had some degree of pectus . Does my risk go done now that the pectus has been repaired for several years ? Thanks
(from: Training for a Heart Attack)
The cardiac tests are done to find out whether you have coronary artery or other heart disease, the most important factors to exclude when new onset PVCs occur. They include blood tests, an echocardiogram and often a cardiac stress test.
- An IVC filter is placed to limit blood clots from below to travel to your heart and lungs and potentially cause a pulmonary embolism, but is otherwise unrelated to a AF and/or the need to perform an ablation.
Electroanatomic map of the posterior left atrium, illustrating the pulmonary veins: right superior pulmonary vein (RSPV), right inferior pulmonary vein (RIPV), left superior pulmonary vein (LSPV), and left inferior pulmonary vein (LIPV). The red circles represent actual discrete radiofrequency applications, predominantly delivered in a circumferential pattern around the pulmonary veins. This ablation strategy can isolate pulmonary vein foci that initiate atrial fibrillation, and/or alter the substrate of the left atrium to inhibit fibrillatory activity due to reentry. Image courtesy of American College of Cardiology Foundation.
Even though I had some techncal problems before and during the presentation, I believe this video session was a worthy first endeavour: